SCPI conducts research into the biologic and physiologic consequences of using aborted fetal material for drug, vaccine and cosmetic production.

Our research focus is on genomic instability created by the presence of residual contaminating short human DNA fragments and viruses.  Drugs and vaccines are too large to produce in a test tube, and therefore, they must be manufactured using cell lines.  The final products contain contaminants from the cell line used to manufacture the drug or vaccine.  When animal cell lines are utilized, these contaminants are recognized by our immune systems as ‘foreign’ and are eliminated from our bodies.  However, when aborted human fetal or other primitive human cell lines are used, these contaminants have the potential to trigger auto-immune diseases or genomic instability.  When we inject our children or ourselves with aborted fetal produced vaccines, and when we use cosmetics made using aborted fetal cells, we are also injecting or transferring DNA and viruses from the aborted fetus that was used to create the manufacturing cell line.

SCPI is applying the extensive bodies of literature from the study of meiotic recombination, DNA repair mechanisms and gene therapy to determine the impact of aborted fetal DNA contaminants on auto-immune responses and on genomic instability.   During meiotic recombination, genetic material from the chromosomes of the father and the mother is ‘exchanged’, so that the offspring have genetic material that is an intermixing of maternal and paternal material, creating a completely unique genetic individual.  Exchange of genetic material during meiotic recombination is a beneficial process, designed to generate diversity in our offspring.  However, inappropriate activation of the proteins involved in meiotic recombination can lead to genomic instability, cancer and other diseases. 

DNA repair is a process that our cells use to correct mistakes that are introduced into our genes on a daily basis, simply by the fact that we use our genes to make proteins and to make new cells during cell division.  When DNA repair goes awry, genomic instability and disease can be the result.